Hensley, K., Howard, B., Carney, J., & Butterfield, D. (1995). Membrane protein alterations in rodent erythrocytes and synaptosomes due to aging and hyperoxia. Biochimica et Biophysica Acta, 1270(2-3), 203-6. https://doi.org/10.1016/0925-4439(95)00043-4 (Original work published 1995)

Mark, R., Hensley, K., Butterfield, D., & Mattson, M. (1995). Amyloid beta-peptide impairs ion-motive ATPase activities: evidence for a role in loss of neuronal Ca2+ homeostasis and cell death. The Journal of Neuroscience : The Official Journal of the Society for Neuroscience, 15(9), 6239-49. Retrieved from http://www.jneurosci.org/cgi/pmidlookup?view=long&pmid=7666206

Aksenov, M., Aksenova, M., Harris, M., Hensley, K., Butterfield, D., & Carney, J. (1995). Enhancement of beta-amyloid peptide A beta(1-40)-mediated neurotoxicity by glutamine synthetase. Journal of Neurochemistry, 65(4), 1899-902. https://doi.org/10.1046/j.1471-4159.1995.65041899.x

Poon, H., Hensley, K., , Merchant, M., Lynn, B., Pierce, W., … Butterfield, D. (2005). Redox proteomics analysis of oxidatively modified proteins in G93A-SOD1 transgenic mice--a model of familial amyotrophic lateral sclerosis. Free Radical Biology & Medicine, 39(4), 453-62. https://doi.org/10.1016/j.freeradbiomed.2005.03.030 (Original work published 2005)

Butterfield, D., Martin, L., Carney, J., & Hensley, K. A beta (25-35) peptide displays H2O2-like reactivity towards aqueous Fe2+, nitroxide spin probes, and synaptosomal membrane proteins. Life Sciences, 58(3), 217-28. https://doi.org/10.1016/0024-3205(95)02279-1 (Original work published 1996)

Aksenov, M., Aksenova, M., Butterfield, D., Hensley, K., Vigo-Pelfrey, C., & Carney, J. (1996). Glutamine synthetase-induced enhancement of beta-amyloid peptide A beta (1-40) neurotoxicity accompanied by abrogation of fibril formation and A beta fragmentation. Journal of Neurochemistry, 66(5), 2050-6. https://doi.org/10.1046/j.1471-4159.1996.66052050.x

Hensley, K., Butterfield, D., Hall, N., Cole, P., Subramaniam, R., Mark, R., … Aksenov, M. (1996). Reactive oxygen species as causal agents in the neurotoxicity of the Alzheimer’s disease-associated amyloid beta peptide. Annals of the New York Academy of Sciences, 786, 120-34. https://doi.org/10.1111/j.1749-6632.1996.tb39057.x (Original work published 1996)

Harris, M., Wang, Y., NW, P. , Jr, Hensley, K., Butterfield, D., & Carney, J. (1996). Amyloid beta peptide (25-35) inhibits Na+-dependent glutamate uptake in rat hippocampal astrocyte cultures. Journal of Neurochemistry, 67(1), 277-86. https://doi.org/10.1046/j.1471-4159.1996.67010277.x

Clerici, W., Hensley, K., DiMartino, D., & Butterfield, D. (1996). Direct detection of ototoxicant-induced reactive oxygen species generation in cochlear explants. Hearing Research, 98(1-2), 116-24. https://doi.org/10.1016/0378-5955(96)00075-5 (Original work published 1996)

Howard, B., Yatin, S., Hensley, K., Allen, K., Kelly, J., Carney, J., & Butterfield, D. (1996). Prevention of hyperoxia-induced alterations in synaptosomal membrane-associated proteins by N-tert-butyl-alpha-phenylnitrone and 4-hydroxy-2,2,6,6-tetramethylpiperidin-1-oxyl (Tempol). Journal of Neurochemistry, 67(5), 2045-50. https://doi.org/10.1046/j.1471-4159.1996.67052045.x